In an argument that I generally lose due being shouted down, the real empirical evidence on second-hand smoke (aka "environmental tobacco smoke" or "ETS") indicates that it is harmless. Even if you are in the constant presence of a smoker, the level of exposure is simply not sufficient to cause any MEDICAL harm (no argument that the stuff is obnoxious and offensive). But the studies that rise to the level of public consciousness generally are merely compilations of previous "studies" that were in themselves statistical nonsense. The EPA and the CDC have done this on several occasions. In short, non-smoking spouses of smokers and non-smoking co-workers of smokers have no greater risk of contracting any diseases that are generally associated with cigarette smoking than anyone else. (I was raised in a household with 7 "chain smokers"). But because this goes against "common knowledge" and current public sentiment, the real data is never reported.
I have linked and copied an abstract of a Real study.
http://www.ncbi.nlm.nih.gov/pubmed/9776 ... t=Abstract
Multicenter case-control study of exposure to environmental tobacco smoke and lung cancer in Europe.
Source
International Agency for Research on Cancer, Lyon, France.
boffetta@iarc.fr
Abstract
BACKGROUND:
An association between exposure to environmental tobacco smoke (ETS) and lung cancer risk has been suggested. To evaluate this possible association better, researchers need more precise estimates of risk, the relative contribution of different sources of ETS, and the effect of ETS exposure on different histologic types of lung cancer. To address these issues, we have conducted a case-control study of lung cancer and exposure to ETS in 12 centers from seven European countries.
METHODS:
A total of 650 patients with lung cancer and 1542 control subjects up to 74 years of age were interviewed about exposure to ETS. Neither case subjects nor control subjects had smoked more than 400 cigarettes in their lifetime.
RESULTS:
ETS exposure during childhood was not associated with an increased risk of lung cancer (odds ratio [OR] for ever exposure = 0.78; 95% confidence interval [CI] = 0.64-0.96). The OR for ever exposure to spousal ETS was 1.16 (95% CI = 0.93-1.44). No clear dose-response relationship could be demonstrated for cumulative spousal ETS exposure. The OR for ever exposure to workplace ETS was 1.17 (95% CI = 0.94-1.45), with possible evidence of increasing risk for increasing duration of exposure. No increase in risk was detected in subjects whose exposure to spousal or workplace ETS ended more than 15 years earlier. Ever exposure to ETS from other sources was not associated with lung cancer risk. Risks from combined exposure to spousal and workplace ETS were higher for squamous cell carcinoma and small-cell carcinoma than for adenocarcinoma, but the differences were not statistically significant.
CONCLUSIONS:
Our results indicate no association between childhood exposure to ETS and lung cancer risk. We did find weak evidence of a dose-response relationship between risk of lung cancer and exposure to spousal and workplace ETS. There was no detectable risk after cessation of exposure.
So the chances of getting sick through smoke exposure through the common wall of a condo? Pretty much nonsense